I discuss the mechanisms of oxidative stress and damage to the previous post, but that the impact of cardiovascular disease (CVD)? Atherosclerotic plaque formation of blood vessels that can build over time enough to open the heart and the blood is not enough to take care of the fabric. This leads to tissue death in the region irrigated by an artery. The best known examples of a heart attack and stroke. It is important to obtain a comprehensive understanding of atherosclerosis (plaque to) understand how oxidative stress can damage and consequences. Atherosclerosis is a multifactorial process and may be affected in different ways.
The first risk factor is high LDL (low density lipoprotein, also known as “bad cholesterol”) is known. We know that for many years to reduce a complete data reduction of LDL-cholesterol of cardiovascular diseases. LDL is an important component of atherosclerotic plaques in the arteries. We see them as low as possible, there are several ways to get it (diet, exercise, weight loss, drugs, supplements, vitamins, alcohol, etc.) to do. Cholesterol is an essential part of our cell wall and LDL-cholesterol, which helps cells. Too much LDL cholesterol is bad, it accumulates and causes damage. We have through different mechanisms. HDL helps remove excess LDL cholesterol – which is a good amount of HDL cholesterol is necessary! This is only the beginning of the LDL role in the pathogenesis of atherosclerosis. Take home is that the number of LDL accumulation in the arterial wall.
When LDL cholesterol is deposited in the artery wall is oxidized by hydrogen peroxide, peroxides or free radicals (see article to explain the oxidation). Is oxidized LDL (ox LDL is called) is in itself toxic to cells and contribute to injuries and infections in the area. Macrophages to respond to injury, and oxLDL as a toxic effect. As a result, macrophages become “foam cells”. Other damage to LDL, such as diabetes are present and lead to the same fate.
Inner lining of blood vessels from endothelial cells and put the foam cells in the circulation, cell activation and humoral immunity (speaking guides you to your doctor to explain anti-inflammatory). Ironically, it is to be protected to safeguard against damage to the arteries, but it will grow.
Oxidative stress is an important mechanism of damage to LDL cholesterol, while there are other possibilities, above. Is it possible to prevent oxidative stress damage and reduce plaque and reduce the risk of cardiovascular diseases such as heart attack and stroke.
There are different types of antioxidants. Vitamin C, vitamin E, beta-carotene, polyphenols and many other plants with nutrients and antioxidants to prevent oxidative stress. Since everyone has other similarities to various factors, oxidative stress, it is important to eat a lot of variety. This is a good diet, no pills “booster” achieved.
Excellent source of many sources of natural antioxidants food nutraceutical research EIRO time. It is, of course, full of food, sustainable source of antioxidants. And “if there is anything to go to the source and the consumption of these products that contain them. We should choose a better source .
The first risk factor is high LDL (low density lipoprotein, also known as “bad cholesterol”) is known. We know that for many years to reduce a complete data reduction of LDL-cholesterol of cardiovascular diseases. LDL is an important component of atherosclerotic plaques in the arteries. We see them as low as possible, there are several ways to get it (diet, exercise, weight loss, drugs, supplements, vitamins, alcohol, etc.) to do. Cholesterol is an essential part of our cell wall and LDL-cholesterol, which helps cells. Too much LDL cholesterol is bad, it accumulates and causes damage. We have through different mechanisms. HDL helps remove excess LDL cholesterol – which is a good amount of HDL cholesterol is necessary! This is only the beginning of the LDL role in the pathogenesis of atherosclerosis. Take home is that the number of LDL accumulation in the arterial wall.
When LDL cholesterol is deposited in the artery wall is oxidized by hydrogen peroxide, peroxides or free radicals (see article to explain the oxidation). Is oxidized LDL (ox LDL is called) is in itself toxic to cells and contribute to injuries and infections in the area. Macrophages to respond to injury, and oxLDL as a toxic effect. As a result, macrophages become “foam cells”. Other damage to LDL, such as diabetes are present and lead to the same fate.
Inner lining of blood vessels from endothelial cells and put the foam cells in the circulation, cell activation and humoral immunity (speaking guides you to your doctor to explain anti-inflammatory). Ironically, it is to be protected to safeguard against damage to the arteries, but it will grow.
Oxidative stress is an important mechanism of damage to LDL cholesterol, while there are other possibilities, above. Is it possible to prevent oxidative stress damage and reduce plaque and reduce the risk of cardiovascular diseases such as heart attack and stroke.
There are different types of antioxidants. Vitamin C, vitamin E, beta-carotene, polyphenols and many other plants with nutrients and antioxidants to prevent oxidative stress. Since everyone has other similarities to various factors, oxidative stress, it is important to eat a lot of variety. This is a good diet, no pills “booster” achieved.
Excellent source of many sources of natural antioxidants food nutraceutical research EIRO time. It is, of course, full of food, sustainable source of antioxidants. And “if there is anything to go to the source and the consumption of these products that contain them. We should choose a better source .
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